Anniversary Reaction


Psychological literature calls it the anniversary reaction and defines it as an individual’s response to unresolved grief resulting from significant losses. The anniversary reaction can involve several days or even weeks of anxiety, anger, nightmares, flashbacks, depression, or fear.

On a more positive note, the anniversary of a disaster or traumatic event also can provide an opportunity for emotional healing.




inflammation of the glomeruli of the kidney; characterized by decreased production of urine and by the presence of blood and protein in the urine.



a neurologic disorder where sustained muscle contractions cause twisting and repetitive movements or abnormal postures.

 The disorder may be hereditary or caused by other factors such as birth-related or other physical trauma, infection, poisoning (e.g., lead poisoning) or reaction to pharmaceutical medications, particularly neuroleptics.

A rain drop to the ocean of time


 It takes so little to remind us, that life is short. It is what we do with the time we have that makes it all worthwhile.


Work like you don’t need the money, love like you’ve never been hurt and dance like no one is watching.

– Randall G Leighton

Dyskinetic syndrome


Abnormal involuntary movements which primarily affect the extremities, trunk, or jaw that occur as a manifestation of an underlying disease process. Conditions which feature recurrent or persistent episodes of dyskinesia as a primary manifestation of disease may be referred to as dyskinesia syndromes

Bone Shattered Conclusion



we have come to a


bone shattered conclusion

that there is nothing else

yet everything

is captured

with beautiful simplicity

in the here and now.



characterized by the presence of a higher than normal level of methemoglobin (metHb) in the blood.

Methemoglobin is a form of hemoglobin that does not bind oxygen. When its concentration is elevated in red blood cells, tissue hypoxia can occur.

Normally, methemoglobin levels are <1%, of total blood hemoglobins.



  1. congenital methemoglobinemia – NADH methemoglobin reductase mutation
  2. acquired methemoglobinemia –
  •  exogenous oxidizing drugs and their metabolites
  • benzovaine
  • dapsone
  • nitrates
  •  may accelerate the rate of formation of methemoglobin up to one-thousandfold, overwhelming the protective enzyme systems and acutely increasing methemoglobin levels.